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・ 2,4-Xylidine
・ 2,5-Bis(hydroxymethyl)furan
・ 2,5-diamino-6-(ribosylamino)-4(3H)-pyrimidinone 5'-phosphate reductase
・ 2,5-Diamino-6-hydroxy-4-(5-phosphoribosylamino)pyrimidine
・ 2,5-Diaminotoluene
・ 2,5-diaminovalerate transaminase
・ 2,5-Dichloroaniline
・ 2,5-didehydrogluconate reductase
・ 2,5-Dihydrofuran
・ 2,5-Dihydroxy-1,4-benzoquinone
・ 2,5-Dihydroxycinnamic acid
・ 2,5-dihydroxypyridine 5,6-dioxygenase
・ 2,5-diketocamphane 1,2-monooxygenase
・ 2,5-Diketopiperazine
・ 2,5-Dimethoxy-4-(2-fluoroethyl)amphetamine
2,5-Dimethoxy-4-amylamphetamine
・ 2,5-Dimethoxy-4-bromoamphetamine
・ 2,5-Dimethoxy-4-butylamphetamine
・ 2,5-Dimethoxy-4-chloroamphetamine
・ 2,5-Dimethoxy-4-ethoxyamphetamine
・ 2,5-Dimethoxy-4-ethylamphetamine
・ 2,5-Dimethoxy-4-fluoroamphetamine
・ 2,5-Dimethoxy-4-iodoamphetamine
・ 2,5-Dimethoxy-4-isopropylamphetamine
・ 2,5-Dimethoxy-4-methylamphetamine
・ 2,5-Dimethoxy-4-nitroamphetamine
・ 2,5-Dimethoxy-4-propylamphetamine
・ 2,5-Dimethoxy-4-trifluoromethylamphetamine
・ 2,5-Dimethoxybenzaldehyde
・ 2,5-Dimethylfuran


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2,5-Dimethoxy-4-amylamphetamine : ウィキペディア英語版
2,5-Dimethoxy-4-amylamphetamine

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Dimethoxy-4-amylamphetamine (DOAM) is a lesser-known psychedelic drug and a substituted amphetamine. DOAM was first synthesized by Alexander Shulgin. In his book ''PiHKAL (Phenethylamines i Have Known And Loved)'', the minimum dosage is listed as 10 mg, and the duration is unknown. DOAM produces a bare threshold and tenseness. As the 4-alkyl chain length is increased from shorter homologues such as DOM, DOET and DOPR which are all potent hallucinogens, the 5-HT2 binding affinity increases, rising to a maximum with the 4-(''n''-hexyl) derivative before falling again with even longer chains, but compounds with chain length longer than ''n''-propyl, or with other bulky groups such as isopropyl, ''t''-butyl or γ-phenylpropyl at the 4- position, fail to substitute for hallucinogens in animals or produce hallucinogenic effects in humans, suggesting these have low efficacy and are thus antagonists or weak partial agonists at the 5-HT2A receptor.
==References==


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