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・ Acetylesterase
・ Acetylfentanyl
・ Acetylferrocene
・ Acetylgalactosaminyl-O-glycosyl-glycoprotein b-1,3-N-acetylglucosaminyltransferase
・ Acetylgalactosaminyl-O-glycosyl-glycoprotein b-1,6-N-acetylglucosaminyltransferase
・ Acetylglutamate kinase
・ Acetylglutamate synthetase
・ Acetylglycinamide chloral hydrate
・ Acetylide
・ Acetylindoxyl oxidase
・ Acetylleucine
・ Acetyllysine
・ Acetylmethadol
・ Acetylmethionine sulfoxide reductase
・ Acetylmorphine
Acetylmorphone
・ Acetylornithinase
・ Acetylornithine deacetylase
・ Acetylornithine transaminase
・ Acetylpropionyl
・ Acetylpropionylmorphine
・ Acetylputrescine deacetylase
・ Acetylpyrazine
・ Acetylpyruvate hydrolase
・ Acetylsalicylate deacetylase
・ Acetylsalicylic acid/dipyridamole
・ Acetylserotonin O-methyltransferase
・ Acetylspermidine deacetylase
・ Acetyltransferase
・ Acetylxylan esterase


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Acetylmorphone : ウィキペディア英語版
Acetylmorphone

Acetylmorphone (Dihydromorphinone acetate) is an opiate analogue that is an acetylated derivative of hydromorphone which was developed in the early 1900s as a potential cough suppressant and analgesic. It is prepared by the acetylation of hydromorphone using either acetyl chloride or acetic anhydride. It was banned internationally in 1930 by the Health Committee of the League of Nations, in order to prevent its sale as an analogue of heroin.〔(Esters of Morphine. UNODC Bulletin on Narcotics, 1953; Issue 2:36-38. )〕
Acetylmorphone is not currently used in medicine, but may have a higher bioavailability than hydromorphone due to its greater lipid solubility, and hence is likely to be more potent than the parent drug, although probably slower acting due to the requirement for deacetylation to the active metabolite hydromorphone. It can be expected to have similar side effects to other opiates, which would include itching, nausea and respiratory depression.
==References==



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