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〕 derived from a proenzyme in the gastrointestinal tract called ]. It reduces food intake, in particular fat intake,〔cite book | last =Fernstrom, etc | first = | authorlink = |author2=John D. Fernstrom |author3=Ricardo Uauy |author4=Pedro Arroyo | title =Nutrition and Brain: 5th Nestle Nutrition Workshop, Mexico City, | publisher =Karger Publishers | year =2000 | location = | url = http://books.google.com/?id=cvk76-ZpmSEC&pg=PA76&dq=Enterostatin | doi = | id = isbn=3-8055-7166-6 | page =76 | isbn =978-3-8055-7166-1 }}〕 when given peripherally or into the brain.〔 ==Function== Enterostatin is created in the intestine by pancreatic procolipase, the other colipase serving as an obligatory cofactor for pancreatic lipase during fat digestion. Enterostatin can be created in the gastric mucosa and the mucosal epithelia in the small intestine. An increased high fat diets will cause the procolipase gene transcription and enterostatin to release into the gastrointestinal lumen. Enterostatin appears in the lymph and circulation after a meal. Enterostatin has been shown to selectively reduce fat intake during a normal meal. The testing has been successful with different species. 抄文引用元・出典: フリー百科事典『 ウィキペディア（Wikipedia）』 ■ウィキペディアで「Enterostatin」の詳細全文を読む スポンサード リンク
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